Search results for " neurosciences"

showing 10 items of 89 documents

GABA and Gap Junctions in the Development of Synchronized Activity in Human Pluripotent Stem Cell-Derived Neural Networks.

2017

The electrical activity of the brain arises from single neurons communicating with each other. However, how single neurons interact during early development to give rise to neural network activity remains poorly understood. We studied the emergence of synchronous neural activity in human pluripotent stem cell (hPSC)-derived neural networks simultaneously on a single-neuron level and network level. The contribution of gamma-aminobutyric acid (GABA) and gap junctions to the development of synchronous activity in hPSC-derived neural networks was studied with GABA agonist and antagonist and by blocking gap junctional communication, respectively. We characterized the dynamics of the network-wide…

0301 basic medicineBiolääketieteet - Biomedicineneural networkstem cell derived neuronslcsh:RC321-57103 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineCalcium imagingPremovement neuronal activityhuman pluripotent stem cellsInduced pluripotent stem celllcsh:Neurosciences. Biological psychiatry. Neuropsychiatrygap junctionsOriginal ResearchArtificial neural networkGABAA receptorChemistrymicroelectrode arrayGap junctionsynchronyDepolarizationMultielectrode arraycalcium imaging030104 developmental biologynervous systemexcitatory GABANeuroscienceNeurotieteet - Neurosciences030217 neurology & neurosurgeryNeuroscienceFrontiers in cellular neuroscience
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Dynamic large-scale network synchronization from perception to action

2018

Sensory-guided actions entail the processing of sensory information, generation of perceptual decisions, and the generation of appropriate actions. Neuronal activity underlying these processes is distributed into sensory, fronto-parietal, and motor brain areas, respectively. How the neuronal processing is coordinated across these brain areas to support functions from perception to action remains unknown. We investigated whether phase synchronization in large-scale networks coordinate these processes. We recorded human cortical activity with magnetoencephalography (MEG) during a task in which weak somatosensory stimuli remained unperceived or were perceived. We then assessed dynamic evolutio…

0301 basic medicineComputer sciencemedia_common.quotation_subjectSomatosensorySensory systemSynchronizationSomatosensory systemlcsh:RC321-57103 medical and health sciences0302 clinical medicineArtificial IntelligencePerceptionMotor systemSynchronization (computer science)medicinePremovement neuronal activitylcsh:Neurosciences. Biological psychiatry. Neuropsychiatrymedia_commonMEGmedicine.diagnostic_testApplied MathematicsGeneral NeuroscienceResearchCommunication3112 NeurosciencesMagnetoencephalographyPhase synchronizationComputer Science Applications030104 developmental biologyActionPerceptionNeuroscience030217 neurology & neurosurgery
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Bumetanide prevents brain trauma-induced depressive-like behavior

2019

AbstractBrain trauma triggers a cascade of deleterious events leading to enhanced incidence of drug resistant epilepsies, depression and cognitive dysfunctions. The underlying mechanisms leading to these alterations are poorly understood and treatment that attenuates those sequels not available. Using controlled-cortical impact (CCI) as experimental model of brain trauma in adult mouse we found a strong suppressive effect of the sodium-potassium-chloride importer (NKCC1) specific antagonist bumetanide on appearance of depression-like behavior. We demonstrate that this alteration in behavior is associated with a block of CCI-induced decrease in parvalbumin-positive interneurons and impairmen…

0301 basic medicineDOWN-REGULATIONpotassium chloride cotransporter 2 (KCC2)[SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyHippocampusUP-REGULATION0302 clinical medicineMedicineCOTRANSPORTER KCC2NEURAL STEM-CELLBrain traumaDepression (differential diagnoses)Original Research0303 health sciencesNeurogenesisDepolarizationNeural stem cell3. Good healthADULT HIPPOCAMPAL NEUROGENESISneurogenesis[SDV.SP.PHARMA] Life Sciences [q-bio]/Pharmaceutical sciences/PharmacologydepressionBumetanidemedicine.druginterneuron cell deathpsychiatric diseaseINHIBITIONbumetanidelcsh:RC321-571Cellular and Molecular Neuroscience03 medical and health sciencesINJURYlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryMolecular Biology030304 developmental biologybusiness.industryMechanism (biology)GRANULE CELLSDentate gyrusAntagonist3112 Neurosciences[SDV.NEU.NB] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Neurobiology030104 developmental biologyDENTATE GYRUSDIURETIC BUMETANIDE[SDV.SP.PHARMA]Life Sciences [q-bio]/Pharmaceutical sciences/PharmacologybusinessNeuroscience030217 neurology & neurosurgeryNeuroscience
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Neurochemical Phenotype of Reelin Immunoreactive Cells in the Piriform Cortex Layer II

2016

Reelin, a glycoprotein expressed by Cajal-Retzius neurons throughout the marginal layer of developing neocortex, has been extensively shown to play an important role during brain development, guiding neuronal migration and detachment from radial glia. During the adult life, however, many studies have associated Reelin expression to enhanced neuronal plasticity. Although its mechanism of action in the adult brain remains mostly unknown, Reelin is expressed mainly by a subset of mature interneurons. Here, we confirm the described phenotype of this subpopulation in the adult neocortex. We show that these mature interneurons, although being in close proximity, lack polysialylated neural cell ad…

0301 basic medicineLow-density lipoprotein receptor-related protein 8PSA-NCAMlcsh:RC321-57103 medical and health sciencesCellular and Molecular Neurosciencepiriform cortex0302 clinical medicineADULT-RATSYNAPTIC PLASTICITYCEREBRAL-CORTEXPiriform cortexmedicineMESSENGER-RNA EXPRESSIONPSA-NCAM EXPRESSIONReelinCajal-Retzius cellslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryOriginal ResearchNeocortexbiology3112 NeurosciencesNONHUMAN-PRIMATESReelinDAB1DoublecortinDOUBLECORTIN-EXPRESSING CELLS030104 developmental biologymedicine.anatomical_structureSTRUCTURAL PLASTICITYnervous systemDCXbiology.proteinNeural cell adhesion moleculeNeuNNeuroscienceHIPPOCAMPAL CONNECTIONS030217 neurology & neurosurgeryNeuroscienceFrontiers in Cellular Neuroscience
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Negatively Charged Gangliosides Promote Membrane Association of Amphipathic Neurotransmitters

2018

Lipophilic neurotransmitters (NTs) such as dopamine are chemical messengers enabling neurotransmission by adhering onto the extracellular surface of the post-synaptic membrane in a synapse, followed by binding to their receptors. Previous studies have shown that the strength of the NT-membrane association is dependent on the lipid composition of the membrane. Negatively charged lipids such as phosphatidylserine, phosphatidylglycerol, and phosphatidic acid have been indicated to promote NT-membrane binding, however these anionic lipids reside almost exclusively in the intracellular leaflet of the post-synaptic membrane instead of the extracellular leaflet facing the synaptic cleft. Meanwhile…

0301 basic medicineMOLECULAR-DYNAMICS SIMULATIONSBIOMOLECULAR SYSTEMSkolesteroliasetyylikoliiniSynaptic TransmissionsolukalvotCell membranechemistry.chemical_compoundSCHIZOPHRENIAmolekyylidynamiikkamolecular dynamics (MD)neurotransmissionvälittäjäaineetChemistryLIPID-MEMBRANESGeneral NeurosciencePhosphatidylserineALZHEIMERS-DISEASEMembranemedicine.anatomical_structureHAMILTONIAN REPLICA EXCHANGElipids (amino acids peptides and proteins)dopamineIntracellularneurotransmittermonosialotetrahexosylganglioside (GM1)Synaptic cleftG(M1) GangliosideMolecular Dynamics SimulationNeurotransmission03 medical and health sciencesExtracellularmedicineAnimalsmonosialotetrahexosylgangliosidebinding free energyPhosphatidylglyceroldopamiiniBinding SitesCell Membranehistamiini3112 Neurosciencesta1182cholesterolBILAYERhistamineacetylcholinehermosolut030104 developmental biologyFORCE-FIELDBiophysicssynapsit
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Passive exposure to speech sounds induces long-term memory representations in the auditory cortex of adult rats

2016

AbstractExperience-induced changes in the functioning of the auditory cortex are prominent in early life, especially during a critical period. Although auditory perceptual learning takes place automatically during this critical period, it is thought to require active training in later life. Previous studies demonstrated rapid changes in single-cell responses of anesthetized adult animals while exposed to sounds presented in a statistical learning paradigm. However, whether passive exposure to sounds can form long-term memory representations remains to be demonstrated. To investigate this issue, we first exposed adult rats to human speech sounds for 3 consecutive days, 12 h/d. Two groups of …

0301 basic medicineMaleSTIMULUS-SPECIFIC ADAPTATIONLong-Term PotentiationMismatch negativityAudiologyMAGNITUDE0302 clinical medicineauditory cortexPLASTICITYBRAINta515media_commonSound (medical instrument)MultidisciplinaryLong-term memorySoundPsychologymedicine.medical_specialtyMISMATCH NEGATIVITY MMN515 Psychologymedia_common.quotation_subjectEVENT-RELATED POTENTIALSFREQUENCYAuditory cortexta3112Article03 medical and health scienceslong-term memoryEvent-related potentialPerceptual learningMemoryPerceptionmedicineotorhinolaryngologic diseasesAnimalsSpeechRats WistarAuditory CortexPERCEPTIONpassive exposure3112 Neuroscienceshuman speechsäilömuistiRatsratsElectrophysiology030104 developmental biologyDISCRIMINATION030217 neurology & neurosurgeryRESPONSESScientific Reports
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Intravenous thrombolysis for suspected ischemic stroke with seizure at onset

2019

Objective Seizure at onset (SaO) has been considered a relative contraindication for intravenous thrombolysis (IVT) in patients with acute ischemic stroke, although this appraisal is not evidence based. Here, we investigated the prognostic significance of SaO in patients treated with IVT for suspected ischemic stroke. Methods In this multicenter, IVT-registry-based study we assessed the association between SaO and symptomatic intracranial hemorrhage (sICH, European Cooperative Acute Stroke Study II definition), 3-month mortality, and 3-month functional outcome on the modified Rankin Scale (mRS) using unadjusted and adjusted logistic regression, coarsened exact matching, and inverse probabil…

0301 basic medicineMalemedicine.medical_specialtymedicine.medical_treatmentLogistic regressionGUIDELINESCLASSIFICATION3124 Neurology and psychiatryBrain IschemiaEARLY MANAGEMENT03 medical and health sciences0302 clinical medicineALTEPLASEModified Rankin ScaleSeizuresMIMICSInternal medicineMedicineHumansThrombolytic TherapyPLASMINOGEN-ACTIVATOR610 Medicine & healthStrokeContraindicationPOPULATIONAgedAged 80 and overHEALTH-CARE PROFESSIONALSbusiness.industryConfounding3112 NeurosciencesOdds ratioThrombolysisASSOCIATIONMiddle Agedmedicine.diseasePrognosisConfidence intervalStroke030104 developmental biologyTreatment OutcomeNeurologySAFETYFemaleNeurology (clinical)business030217 neurology & neurosurgery
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Surge of Peripheral Arginine Vasopressin in a Rat Model of Birth Asphyxia

2018

Mammalian birth is accompanied by a period of obligatory asphyxia, which consists of hypoxia (drop in blood O2 levels) and hypercapnia (elevation of blood CO2 levels). Prolonged, complicated birth can extend the asphyxic period, leading to a pathophysiological situation, and in humans, to the diagnosis of clinical birth asphyxia, the main cause of hypoxic-ischemic encephalopathy (HIE). The neuroendocrine component of birth asphyxia, in particular the increase in circulating levels of arginine vasopressin (AVP), has been extensively studied in humans. Here we show for the first time that normal rat birth is also accompanied by an AVP surge, and that the fetal AVP surge is further enhanced in…

0301 basic medicineVasopressinmedicine.medical_specialtySTRESSArgininehypothalamic-pituitary axis (HPA axis)blood gasesHYPOXIAbirth asphyxia3124 Neurology and psychiatrylcsh:RC321-571neonatal03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineCopeptinInternal medicineMedicineBRAINlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryPRECURSORNEURONSperinatalOriginal ResearchRELEASEAsphyxiaFetusPARAVENTRICULAR NUCLEUSbusiness.industry3112 NeurosciencescopeptinENCEPHALOPATHYarginine vasopressin (AVP)Hypoxia (medical)base deficit030104 developmental biologyEndocrinologyHypothalamusHYPOTHALAMUSmedicine.symptombusinessHypercapnia030217 neurology & neurosurgeryNeuroscienceFrontiers in Cellular Neuroscience
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Effects of PSA Removal from NCAM on the Critical Period Plasticity Triggered by the Antidepressant Fluoxetine in the Visual Cortex.

2016

Neuronal plasticity peaks during critical periods of postnatal development and is reduced towards adulthood. Recent data suggests that windows of juvenile-like plasticity can be triggered in the adult brain by antidepressant drugs such as Fluoxetine. Although the exact mechanisms of how Fluoxetine promotes such plasticity remains unknown, several studies indicate that inhibitory circuits play an important role. The polysialylated form of the neural cell adhesion molecules (PSA-NCAM) has been suggested to mediate the effects of Fluoxetine and it is expressed in the adult brain by mature interneurons. Moreover, the enzymatic removal of PSA by neuroaminidase-N not only affects the structure of…

0301 basic medicinegenetic structuresPSA-NCAMta3112lcsh:RC321-571critical period plasticity03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineparvalbumin interneuronsSYNAPTIC PLASTICITYNeuroplasticitymedicinevisual plasticityMONOCULAR DEPRIVATIONlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryREGULATES PLASTICITYOriginal ResearchbiologyMEDIAL PREFRONTAL CORTEXPOLYSIALIC ACID3112 NeurosciencesCELLULAR AND MOLECULAR NEUROSCIENCEfluoxetineLong-term potentiationSciences bio-médicales et agricoles3. Good healthOCULAR DOMINANCE PLASTICITYMonocular deprivation030104 developmental biologyVisual cortexmedicine.anatomical_structureSTRUCTURAL PLASTICITYnervous systemCELL-ADHESION MOLECULESynaptic plasticitybiology.proteinNeural cell adhesion moleculeLONG-TERM POTENTIATIONPsychologyNeuroscience030217 neurology & neurosurgeryParvalbuminNeuroscienceNEUROTROPHIC FACTORFOSB
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Matlab Open Source Code: Noise-Assisted Multivariate Empirical Mode Decomposition Based Causal Decomposition for Causality Inference of Bivariate Tim…

2022

Causality inference has arrested much attention in academic studies. Currently, multiple methods such as Granger causality, Convergent Cross Mapping (CCM), and Noise-assisted Multivariate Empirical Mode Decomposition (NA-MEMD) are introduced to solve the problem. Motivated by the researchers who uploaded the open-source code for causality inference, we hereby present the Matlab code of NA-MEMD Causal Decomposition to help users implement the algorithm in multiple scenarios. The code is developed on Matlab2020 and is mainly divided into three subfunctions: na_memd, Plseries, and cd_na_memd. na_memd is called in the main function to generate the matrix of Intrinsic Mode Functions (IMFs) and P…

1109 Neurosciences 1702 Cognitive SciencesBiomedical EngineeringNeuroscience (miscellaneous)Computer Science Applications
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